Hair concern
Hair Thinning
Hair thinning can stem from genetics, hormonal change, nutritional deficiency or follicle-level inflammation. Clinical assessment determines which mechanism is driving the loss, and which approach is most appropriate.
Understanding the cause
What's happening in your skin
Something disrupted your hair cycle
In telogen effluvium, shedding appears weeks or months after the trigger, which is why most people have been losing density longer than they realise.
Every follicle cycles through anagen (active growth), catagen (transition) and telogen (rest, then shed). In androgenetic alopecia, DHT miniaturises follicles progressively until they go quiet. In telogen effluvium, an unusually high proportion of follicles enter telogen after a triggering event. Alopecia areata involves immune-mediated follicle damage. Each pattern looks different and responds to different treatment.
Act before follicles go quiet
Once a follicle has been dormant long enough, its capacity to respond to treatment diminishes. The window matters.
Clinical treatments work most effectively on follicles that retain biological activity. People often delay because the change is gradual or because supplements promise improvement without delivering it. By the time most clients arrive, they have been losing density longer than they realise. Identifying the cause quickly is the priority.
Restoring the follicle environment
PRP delivers growth factors PDGF, VEGF and IGF-1 directly to the scalp, extending the anagen growth phase in active follicles.
PRP and polynucleotides deliver concentrated growth factors to the scalp, stimulating dormant follicles and extending anagen. Exosome therapy reduces follicular inflammation and improves the scalp microenvironment. Clinical assessment comes first. The treatment pathway is built around what is driving the thinning.
