Hair

Hair Loss and Thinning: When to Seek Help and What to Expect

Hair thinning affects more people than openly discuss it. Clinic Director Lydia Griffin explains the most common types, what drives them, when in-clinic treatment is worth considering, and what realistic outcomes look like.

Published 21 May 2026


Most people who are losing hair notice it privately for months before mentioning it to anyone. A widening parting, more hair on the pillow, a ponytail that feels thinner, a hairline that looks different. The reluctance to raise it is understandable. What helps is knowing what is actually happening and whether there is anything worth doing about it.

This article covers the most common types of hair loss seen in aesthetic clinic patients, what drives them, when in-clinic treatment makes sense, and what it can realistically achieve.


Understanding the hair growth cycle

Every hair follicle moves independently through a cycle of three phases. Anagen is the active growth phase, lasting two to seven years. Catagen is a brief transitional phase. Telogen is the resting phase, lasting around three months, at the end of which the hair sheds and the follicle restarts. On a healthy scalp, around 85-90% of follicles are in anagen at any one time. The average person sheds 50-100 hairs per day as part of this normal cycle.

Hair loss becomes pathological when this balance shifts: too many follicles enter telogen simultaneously, follicles miniaturise over time and produce progressively finer hair, or follicles stop cycling altogether.


The most common types

Androgenetic alopecia is the most prevalent form, affecting a significant proportion of both men and women by midlife. In men it follows a recognisable pattern. In women the pattern is typically more diffuse: a widening central parting, generalised thinning across the top of the scalp, with the frontal hairline usually preserved. The underlying mechanism is the sensitivity of certain follicles to dihydrotestosterone (DHT). Androgenetic alopecia is genetic and progressive. It can be slowed and density supported, but it is not reversed.

Telogen effluvium is diffuse shedding triggered by a systemic event that shifts a large number of follicles into the resting phase simultaneously. Common triggers include significant illness or fever, major surgery, crash dieting, severe emotional stress, childbirth and thyroid dysfunction. In most cases where the trigger resolves, the shedding is self-limiting and hair density recovers over six to twelve months.

Nutritional deficiency. Ferritin (stored iron), vitamin D, zinc and B vitamins all play a role in healthy hair cycling. Bloodwork before beginning any treatment course is an important step.

Alopecia areata is an autoimmune condition that produces patchy hair loss. It requires dermatology input and is outside the scope of aesthetic scalp treatment. If the presentation suggests alopecia areata, we say so and refer.

Traction alopecia results from chronic tension on the follicle from tight hairstyles. It is progressive if the traction continues and partially reversible if caught early.


Before any in-clinic treatment: rule out the addressable causes

The most useful first step in hair loss is a proper assessment that establishes what is actually driving the change. This means a detailed history and, in most cases, basic blood tests covering ferritin, full blood count, thyroid function, vitamin D and zinc.

Where a nutritional deficiency or thyroid dysfunction is identified, addressing that directly produces better results than in-clinic scalp treatment alone.


When in-clinic treatment helps

For patients with androgenetic alopecia in its early to moderate stages, in-clinic treatment can meaningfully slow the progression, improve scalp environment and in some cases support a modest recovery in density. What it cannot do is stop androgenetic alopecia in the way that medical treatments (finasteride, minoxidil) can.

Scalp mesotherapy with microneedling combines controlled micro-channels in the scalp with targeted serums designed to improve follicular circulation, nutrient delivery and scalp environment.

PRP and iPRF for scalp introduce concentrated growth factors or a sustained-release fibrin matrix directly into the scalp tissue. The evidence base for PRP in hair loss is growing, with multiple published studies showing improvements in hair count and density in androgenetic alopecia.

Exosome therapy for scalp using BLESKIN EXXO applies cell-derived signalling vesicles to the scalp following microneedling, supporting the repair and renewal environment around the follicle.

Polynucleotides for scalp are increasingly used in hair restoration protocols for their anti-inflammatory properties and support of tissue repair.

Following an in-person consultation with our prescribing clinician, in line with current GMC, NMC, GPhC and GDC guidance.


What to expect

Hair treatment courses require patience. The hair growth cycle means that any in-clinic stimulus takes months to produce visible results, typically three to six months before meaningful change in density is apparent. A course of treatment is generally six sessions or more, and results are maintained rather than permanent.


Frequently asked questions

How do I know if I am losing more hair than normal?
The average person sheds 50-100 hairs per day through normal cycling. If you are noticing significantly more than that on the pillow, in the shower or on a brush, or if you can see a visible change in your parting, density or hairline, it is worth seeking assessment. A progressive change over months is worth investigating.
Should I get blood tests before starting a hair treatment course?
Yes, in most cases. Ferritin in particular is frequently overlooked and is a common driver of hair thinning in women. Treating the scalp while a nutritional deficiency remains unaddressed produces limited results. We recommend basic bloodwork covering ferritin, full blood count, thyroid function and vitamin D before committing to a treatment course.
Can PRP regrow hair that has already been lost?
PRP can support follicles that are miniaturised but still active, potentially slowing miniaturisation and in some cases modestly improving density. It cannot regenerate follicles that have been permanently lost over many years. The earlier treatment begins in the course of androgenetic alopecia, the more follicular activity remains to support.
Is hair loss treatment different for women than men?
The pattern, presentation and underlying driver are often different. Female androgenetic alopecia typically presents as diffuse thinning across the crown. The hormonal context differs, and the relevance of oestrogen, iron and thyroid is higher in women on average. The in-clinic treatments are the same, but the accompanying assessment and lifestyle conversation differ between patients.
Can tight hairstyles cause permanent hair loss?
Yes, if traction continues over a sustained period. Traction alopecia tends to begin at the hairline and temples where tension is greatest. In its early stages it is largely reversible when the traction is removed. In long-standing cases where follicular scarring has occurred, the loss in affected areas may be permanent.
How often would I need maintenance sessions after a course?
For most patients with androgenetic alopecia, maintenance of one to two sessions per year is realistic to sustain the improvement from an initial course. The exact interval depends on how the scalp is responding, whether medical treatment is being used alongside, and the progression rate of the underlying condition.

Related advice

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Following an in-person consultation with our prescribing clinician, in line with current GMC, NMC, GPhC and GDC guidance.

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